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Hydrogen peroxide–induced gene expression in Arabidopsis thaliana - ScienceDirect
Antioxidants | Free Full-Text | Nox, Reactive Oxygen Species and Regulation of Vascular Cell Fate
Subtropical Freshwater Cyanobacterial Blooms as Hydrogen Peroxide Hot Spots | Environmental Science & Technology Letters
Elicitor-stimulated ion fluxes and O2− from the oxidative burst are essential components in triggering defense gene activation and phytoalexin synthesis in parsley | PNAS
Subtropical Freshwater Cyanobacterial Blooms as Hydrogen Peroxide Hot Spots | Environmental Science & Technology Letters
Review of the current status of tooth whitening with the ... - Oral Way
Transient Exposure to Hydrogen Peroxide Causes an Increase in Mitochondria-Derived Superoxide As a Result of Sustained Alteration in L-Type Ca2+ Channel Function in the Absence of Apoptosis in Ventricular Myocytes | Circulation Research
Enzymatic reactions towards aldehydes: An overview - Schober - Flavour and Fragrance Journal - Wiley Online Library
Nox4: A Hydrogen Peroxide-Generating Oxygen Sensor | Biochemistry
Plants | Free Full-Text | Electrical Signals, Plant Tolerance to Actions of Stressors, and Programmed Cell Death: Is Interaction Possible?
Thioether Sulfur Oxygenation from O2 or H2O2 Reactivity of Copper Complexes with Tridentate N2Sthioether Ligands | Inorganic Chemistry
Time-Course Diffusion of Hydrogen Peroxide Through Human Dentin: Clinical Significance for Young Tooth Internal Bleaching
Spatial H2O2 Signaling Specificity: H2O2 from Chloroplasts and Peroxisomes Modulates the Plant Transcriptome Differentially - ScienceDirect
Peroxide resistance of ER Ca2+ pump in endothelium: implications to coronary artery function | American Journal of Physiology-Cell Physiology
Principle unified reaction mechanism of peroxide detoxification by... | Download Scientific Diagram
Nox4: A Hydrogen Peroxide-Generating Oxygen Sensor | Biochemistry
Transient Exposure to Hydrogen Peroxide Causes an Increase in Mitochondria-Derived Superoxide As a Result of Sustained Alteration in L-Type Ca2+ Channel Function in the Absence of Apoptosis in Ventricular Myocytes | Circulation Research
Responses of marine macroalgae to hydrogen-peroxide stress - ScienceDirect
Transient Exposure to Hydrogen Peroxide Causes an Increase in Mitochondria-Derived Superoxide As a Result of Sustained Alteration in L-Type Ca2+ Channel Function in the Absence of Apoptosis in Ventricular Myocytes | Circulation Research
Induction of H2O2 in transgenic rice leads to cell death and enhanced resistance to both bacterial and fungal pathogens
Transient Exposure to Hydrogen Peroxide Causes an Increase in Mitochondria-Derived Superoxide As a Result of Sustained Alteration in L-Type Ca2+ Channel Function in the Absence of Apoptosis in Ventricular Myocytes | Circulation Research
